Photo of Wei Li
Contact Expert

View Scopus Profile

Wei Li, PhD

 https://orcid.org/0000-0001-5696-4309
    • 912 Citations
    • 12 h-Index
    20062023

    Research output per year

    If you made any changes in Pure these will be visible here soon.

    Personal profile

    Research interests

    Dr. Wei Li’s laboratory is interested in studying intercellular interactions during tissue homeostasis and determining how de-regulated interactions among cells promote cell transformation and tumor progression. His research integrates molecular, cellular and biochemical approaches in combination with mouse tumorigenesis models. Currently, Dr. Li’s laboratory focuses on elucidating the regulation and function of the Hippo tumor suppressor pathway in brain tumors. The goal of Dr. Li’s research is to find molecular targets and biomarkers for cancer therapy.

    Fingerprint Dive into the research topics where Wei Li is active. These topic labels come from the works of this person. Together they form a unique fingerprint.

    • 2 Similar Profiles

    Network Recent external collaboration on country level. Dive into details by clicking on the dots.

    Projects

    THE ROLE OF NONO IN TAZ-DRIVEN GLIOMA MALIGNANT TRANSFORMATION

    Li, W.

    9/1/18 → 7/31/23

    Project: Research project

    Research Output

    • 912 Citations
    • 12 h-Index
    • 15 Article
    • 4 Review article

    Differential YAP expression in glioma cells induces cell competition and promotes tumorigenesis

    Liu, Z., Yee, P. P., Wei, Y., Liu, Z., Imamura, Y. & Li, W., Mar 1 2019, In : Journal of Cell Science. 132, 5, jcs225714.

    Research output: Contribution to journal › Article

    Open Access
    • 5 Scopus citations

    Induction of store-operated calcium entry (SOCE) suppresses glioblastoma growth by inhibiting the Hippo pathway transcriptional coactivators YAP/TAZ

    Liu, Z., Wei, Y., Zhang, L., Yee, P. P., Johnson, M., Zhang, X., Gulley, M., Atkinson, J. M., Trebak, M., Wang, H-G. & Li, W., Jan 3 2019, In : Oncogene. 38, 1, p. 120-139 20 p.

    Research output: Contribution to journal › Article

    • 9 Scopus citations

    Inhibition of TAZ contributes radiation-induced senescence and growth arrest in glioma cells

    Zhang, L., Cheng, F., Wei, Y., Zhang, L., Guo, D., Wang, B. & Li, W., Apr 11 2019, In : Oncogene. 38, 15, p. 2788-2799 12 p.

    Research output: Contribution to journal › Article

    • 7 Scopus citations

    YAP1 subgroup supratentorial ependymoma requires TEAD and nuclear factor I-mediated transcriptional programmes for tumorigenesis

    Pajtler, K. W., Wei, Y., Okonechnikov, K., Silva, P. B. G., Vouri, M., Zhang, L., Brabetz, S., Sieber, L., Gulley, M., Mauermann, M., Wedig, T., Mack, N., Imamura Kawasawa, Y., Sharma, T., Zuckermann, M., Andreiuolo, F., Holland, E., Maass, K., Körkel-Qu, H., Liu, H. K. & 13 others, Sahm, F., Capper, D., Bunt, J., Richards, L. J., Jones, D. T. W., Korshunov, A., Chavez, L., Lichter, P., Hoshino, M., Pfister, S. M., Kool, M., Li, W. & Kawauchi, D., Dec 1 2019, In : Nature communications. 10, 1, 3914.

    Research output: Contribution to journal › Article

    Open Access

    • 2016 Children's Tumor Foundation conference on neurofibromatosis type 1, neurofibromatosis type 2, and schwannomatosis

    Fisher, M. J., Belzberg, A. J., de Blank, P., De Raedt, T., Elefteriou, F., Ferner, R. E., Giovannini, M., Harris, G. J., Kalamarides, M., Karajannis, M. A., Kim, A. R., Lázaro, C., Le, L. Q., Li, W., Listernick, R., Martin, S., Morrison, H., Pasmant, E., Ratner, N., Schorry, E. & 7 others, Ullrich, N. J., Viskochil, D., Weiss, B., Widemann, B. C., Zhu, Y., Bakker, A. & Serra, E., May 2018, In : American Journal of Medical Genetics, Part A. 176, 5, p. 1258-1269 12 p.

    Research output: Contribution to journal › Article

    • 2 Scopus citations