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Personal profile

Research interests

Dr. Wei Li’s laboratory is interested in studying intercellular interactions during tissue homeostasis and determining how de-regulated interactions among cells promote cell transformation and tumor progression. His research integrates molecular, cellular and biochemical approaches in combination with mouse tumorigenesis models. Currently, Dr. Li’s laboratory focuses on elucidating the regulation and function of the Hippo tumor suppressor pathway in brain tumors. The goal of Dr. Li’s research is to find molecular targets and biomarkers for cancer therapy.

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  • 1 Similar Profiles
Neurofibromin 2 Medicine & Life Sciences
Ubiquitin-Protein Ligases Medicine & Life Sciences
Neoplasms Medicine & Life Sciences
Carcinogenesis Medicine & Life Sciences
Neurofibromatosis 2 Medicine & Life Sciences
Genes Chemical Compounds
Cells Chemical Compounds
Tumors Chemical Compounds

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Projects 2018 2023

Research Output 2006 2019

  • 883 Citations
  • 12 h-Index
  • 14 Article
  • 4 Review article
4 Citations (Scopus)

Differential YAP expression in glioma cells induces cell competition and promotes tumorigenesis

Liu, Z., Yee, P. P., Wei, Y., Liu, Z., Imamura, Y. & Li, W., Mar 1 2019, In : Journal of Cell Science. 132, 5, jcs225714.

Research output: Contribution to journalArticle

Open Access
Glioma
Carcinogenesis
Neoplasms
Glioblastoma
Growth
6 Citations (Scopus)

Induction of store-operated calcium entry (SOCE) suppresses glioblastoma growth by inhibiting the Hippo pathway transcriptional coactivators YAP/TAZ

Liu, Z., Wei, Y., Zhang, L., Yee, P. P., Johnson, M., Zhang, X., Gulley, M., Atkinson, J. M., Trebak, M., Wang, H-G. & Li, W., Jan 3 2019, In : Oncogene. 38, 1, p. 120-139 20 p.

Research output: Contribution to journalArticle

Protein Kinase C beta
Glioblastoma
Amlodipine
Calcium
Growth
4 Citations (Scopus)

Inhibition of TAZ contributes radiation-induced senescence and growth arrest in glioma cells

Zhang, L., Cheng, F., Wei, Y., Zhang, L., Guo, D., Wang, B. & Li, W., Apr 11 2019, In : Oncogene. 38, 15, p. 2788-2799 12 p.

Research output: Contribution to journalArticle

Glioma
Radiation
Catenins
Growth
Glioblastoma
2 Citations (Scopus)

2016 Children's Tumor Foundation conference on neurofibromatosis type 1, neurofibromatosis type 2, and schwannomatosis

Fisher, M. J., Belzberg, A. J., de Blank, P., De Raedt, T., Elefteriou, F., Ferner, R. E., Giovannini, M., Harris, G. J., Kalamarides, M., Karajannis, M. A., Kim, A. R., Lázaro, C., Le, L. Q., Li, W., Listernick, R., Martin, S., Morrison, H., Pasmant, E., Ratner, N., Schorry, E. & 7 others, Ullrich, N. J., Viskochil, D., Weiss, B., Widemann, B. C., Zhu, Y., Bakker, A. & Serra, E., May 1 2018, In : American Journal of Medical Genetics, Part A. 176, 5, p. 1258-1269 12 p.

Research output: Contribution to journalArticle

Neurofibromatosis 2
Neurofibromatosis 1
Neurofibromatoses
Neurilemmoma
Plexiform Neurofibroma
35 Citations (Scopus)

Molecular analysis of aggressive renal cell carcinoma with unclassified histology reveals distinct subsets

Chen, Y. B., Xu, J., Skanderup, A. J., Dong, Y., Brannon, A. R., Wang, L., Won, H. H., Wang, P. I., Nanjangud, G. J., Jungbluth, A. A., Li, W., Ojeda, V., Hakimi, A. A., Voss, M. H., Schultz, N., Motzer, R. J., Russo, P., Cheng, E. H., Giancotti, F. G., Lee, W. & 4 others, Berger, M. F., Tickoo, S. K., Reuter, V. E. & Hsieh, J. J., Oct 7 2016, In : Nature communications. 7, 13131.

Research output: Contribution to journalArticle

Histology
histology
mutations
Renal Cell Carcinoma
set theory