Cardiovascular Physiology in Obesity

Eric J. Chan, Martin A. Alpert

Research output: Chapter in Book/Report/Conference proceedingChapter

3 Scopus citations

Abstract

Obesity produces a variety of hemodynamic changes that cause alterations in cardiac morphology and ventricular function. These alterations may be intensified by the presence of systemic hypertension. In those with severe obesity these pathophysiologic sequelae may contribute to the development of left ventricular failure, pulmonary arterial hypertension, and subsequent right ventricular failure. Sleep apnea and obesity hypoventilation may help to promote pulmonary arterial hypertension and further facilitate right heart decompensation. Such patients are at particularly high risk for these complications in the critical care setting. Knowledge of cardiovascular pathophysiology in obesity may assist the intensivist in the successful management of these challenging patients.

Original languageEnglish (US)
Title of host publicationCritical Care Management of the Obese Patient
PublisherWiley-Blackwell
Pages1-12
Number of pages12
ISBN (Print)9780470655900
DOIs
StatePublished - Apr 19 2012

All Science Journal Classification (ASJC) codes

  • Medicine(all)

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