Choroidal nonperfusion in giant cell arteritis

David Quillen, William Cantore, S. R. Schwartz, R. D. Brod, Joseph Sassani

Research output: Contribution to journalArticle

24 Citations (Scopus)

Abstract

A 68-year-old man had visual loss secondary to isolated choroidal nonperfusion as a clinical manifestation of giant cell arteritis. Ophthalmoscopy disclosed scattered yellow-white lesions at the level of the retinal pigment epithelium in the posterior pole of the right eye. Intravenous fluorescein angiography demonstrated marked delay in choroidal filling of the macula in the right eye. There was no ophthalmoscopic or angiographic evidence of anterior ischemic optic neuropathy or central retinal artery occlusion. After approximately 72 hours of intravenous corticosteroid therapy, the patient's visual acuity improved and repeat intravenous fluorescein angiography showed normal choroidal circulation. Isolated choroidal ischemia is a potential cause of reversible visual loss in patients with giant cell arteritis.

Original languageEnglish (US)
Pages (from-to)171-175
Number of pages5
JournalAmerican Journal of Ophthalmology
Volume116
Issue number2
DOIs
StatePublished - Jan 1 1993

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Giant Cell Arteritis
Fluorescein Angiography
Patient Acuity
Retinal Artery Occlusion
Ischemic Optic Neuropathy
Ophthalmoscopy
Retinal Pigment Epithelium
Visual Acuity
Adrenal Cortex Hormones
Ischemia
Therapeutics

All Science Journal Classification (ASJC) codes

  • Ophthalmology

Cite this

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Choroidal nonperfusion in giant cell arteritis. / Quillen, David; Cantore, William; Schwartz, S. R.; Brod, R. D.; Sassani, Joseph.

In: American Journal of Ophthalmology, Vol. 116, No. 2, 01.01.1993, p. 171-175.

Research output: Contribution to journalArticle

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